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Chilean Journal of Agricultural Research
Instituto de Investigaciones Agropecuarias, INIA
ISSN: 0718-5820
EISSN: 0718-5839
Vol. 77, No. 2, 2017, pp. 150-154
Bioline Code: cj17018
Full paper language: English
Document type: Research Article
Document available free of charge

Chilean Journal of Agricultural Research, Vol. 77, No. 2, 2017, pp. 150-154

 en Resistance level and target-site mechanism to fenoxaprop-p-ethyl in Beckmannia syzigachne check for this species in other resources (Steud.) Fernald populations from China
Li, Lingxu; Luo, Xiaoyong & Wang, Jinxin

Abstract

Beckmannia syzigachne (Steud.) Fernald is one of the main grass weeds severely harming wheat ( Triticum aestivum check for this species in other resources L.) production in rice-wheat areas in China. Fenoxaprop-p-ethyl is the main herbicide used to selectively control grass weed in China. Beckmannia syzigachne has evolved resistance to fenoxaprop-p-ethyl due to continuous application. To investigate fenoxaprop-p- ethyl resistant level and mechanism in B. syzigachne in a portion of the rice-wheat area in China, samples from 31 field populations were collected and treated with fenoxaprop-p-ethyl. The results show that 10 of the 31 tested field populations evolved a high level of resistance to fenoxaprop-p-ethyl. A portion of the acetyl-coenzyme A carboxylase (ACCase) gene was amplified, sequenced and aligned. The known Ile-1781-Leu, Ile-1781-Val, Ile- 2041-Asn, Asp-2078-Gly and Gly-2096-Ala mutations were identified in five resistant populations. None of the known resistant substitutions was identified in the other five resistant populations, which means the resistance to fenoxaprop-p-ethyl in these populations is likely endowed by non-target-site resistance mechanism.

Keywords
ACCase; American slough grass; amino acid substitution; resistant level; resistant mechanism.

 
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