Iranian Journal of Pediatrics
Tehran University of Medical Sciences Press
Vol. 23, No. 1, 2013, pp. 85-88
Bioline Code: pe13014
Full paper language: English
Document type: Short Communication
Document available free of charge
Iranian Journal of Pediatrics, Vol. 23, No. 1, 2013, pp. 85-88
© Copyright 2013 - Iran Journal of Pediatrics
Cardiac Troponin T Levels of Umbilical Cord in Neonates with Abnormal Fetal|
Rafati, Shiva; Rabi, Maryam & Borna, Hajie
Objective: Most neonatal encephalopathic disorders appear to be caused by perinatal events. Persistent
myocardial ischemia leads to cellular necrosis and release of troponin from cardiac muscles. Fetal distress
during labor may be detected by monitoring the fetal heart rate. However little is known about the
relationship, if any, that exists between fetal heart rate abnormalities and the fetal cardiac musculature and its
function. The aim of this study was to investigate the relationship, if any, of umbilical cord serum levels of
cardiac troponin T with fetal bradycardia or late deceleration.
Methods: In this cross sectional study, troponin T level in umbilical cord blood of 80 neonates are measured.
There were 23 versus 57 fetuses with and without late deceleration or bradycardia.
Findings: Level of cardiac troponin T in umbilical blood of neonates with fetal bradycardia or late
deceleration was elevated in comparison to neonates without bradycardia or late deceleration. There was no
relation between umbilical troponin T level and mode of delivery.
Conclusion: Infants with fetal bradycardia or late deceleration during labor had significantly higher cord
cardiac troponin T levels. If troponin level is normal, the probability of hypoxia will be very low.
Umbilical Cord; Cardiac Troponin T; Fetal bradycardia; Late Deceleration
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